stacks



chemotherapy

Cancer, Chemo, and Crony Capitalism

30-day mortality after systemic anticancer treatment

Chemotherapy warning as hundreds die from cancer-fighting drugs

Chemotherapy may spread cancer and trigger aggressive tumors

Publisher retracting more than 100 studies from cancer journal over fake peer reviews

The Secret History Of The War On Cancer


chemotherapy

Phony peer review: The more we look, the more we find



"Breast cancer is the most common indication for chemotherapy among women in the United States, and chemotherapy drugs are the leading cause of serious drug-related adverse effects among women with breast cancer." - Michael J. Hassett, A. James O'Malley, Juliana R. Pakes, Joseph P. Newhouse, and Craig C. Earle

1885 Paul Ehrlich, a German physician and scientist working in the fields of hematology, immunology, and antimicrobial chemotherapy develops the first chemical to target a specific pathogen.

Ehrlich developed the precursor technique to Gram staining bacteria making it possible to distinguish between different types of blood cells leading to the capability of diagnosing numerous blood diseases.

His laboratory discovered arsphenamine (Salvarsan), the first effective medicinal treatment for syphilis, thereby initiating and also naming the concept of chemotherapy.

Paul Ehrlich popularizes the concept of 'a magic bullet'.

Ehrlich reasoned that if a compound could be made that selectively targeted a disease-causing organism, then a toxin - a "magic bullet" - for that organism could be selectivity delivered.

Ehrlich elaborated the systematic testing of chemical compounds as now practiced in the pharmaceutical industry in clinical trials.

Ehrlich's work illuminated the existence of the blood-brain barrier.

1942 Two pharmacologists from the Yale School of Medicine, Louis S. Goodman and Alfred Gilman, are recruited by the DoD to create a less volatile mustard gas.

They exchanged a nitrogen molecule for sulfur creating a more stable compound in nitrogen mustard.

1943 A German air raid in Bari, Italy leads to the exposure of more than a thousand people to a secret cargo of mustard gas bombs on the SS John Harvey.

Autopsies of the victims suggest that profound lymphoid and myeloid suppression occurred after exposure.

Dr. Stewart Francis Alexander theorizes that since mustard gas all but ceased the division of certain types of somatic cells whose nature was to divide fast, it could also potentially be put to use in helping to suppress the division of certain types of cancerous cells.

The British and US governments cover up the presence of mustard gas and its effects on victims of the raid.

Mustard Gas disaster in Bari harbour

"Many oncologists take it for granted that response to therapy prolongs survival, an opinion which is based on a fallacy and which is not supported by clinical studies." - Ulrich Abel



chemothearpy or radiation ?

29% of cancer studies report conflict of interest

More Doctors Confess to Making Cancer Diagnoses Just for Profit

Prominent Michigan Cancer Doctor Pleads Guilty:
'I Knew That It Was Medically Unnecessary'



carmustine, cisplatin, cytarabine

Carmustine is a mustard gas-related compound;

Carmustine, also called BCNU (1,3[bis]-2-chloroethyl-nitrosourea), decomposes spontaneously into a chloroethyl hydroxide that can alkylate the DNA and into an isocyanide molecule, which may produce carbamylation of proteins. Cytotoxicity is caused by DNA cross-links.
Cisplatin is a platinum-based compound;

Cisplatin, mode of action has been linked to its ability to crosslink with the purine bases on the DNA; interfering with DNA repair mechanisms, causing DNA damage, and subsequently inducing apoptosis in cancer cells.

Cytarabine is an antimetabolite antineoplastic agent that inhibits the synthesis of DNA with immunosuppressant properties.

Cytarabine is metabolized intracellularly into its active triphosphate form (cytosine arabinoside triphosphate). Cytosine arabinoside interferes with the synthesis of DNA. Its mode of action is due to its rapid conversion into cytosine arabinoside triphosphate, which damages DNA when the cell cycle holds in the S phase (synthesis of DNA). Rapidly dividing cells, which require DNA replication for mitosis, are therefore most affected. Cytosine arabinoside also inhibits both DNA and RNA polymerases and nucleotide reductase enzymes needed for DNA synthesis.


Cancer Treatment-Induced Neurotoxicity

Hodgkin Lymphoma Treatment Regimens

Cancer therapy selection, dosing, administration, and the management of related adverse events can be a complex process that should be handled by an experienced health care team. Clinicians must choose and verify treatment options based on the individual patient; drug dose modifications and supportive care interventions should be administered accordingly. The cancer treatment regimens below may include both U.S. Food and Drug Administration-approved and unapproved indications/regimens. These regimens are provided only to supplement the latest treatment strategies. These Guidelines are a work in progress that may be refined as often as new significant data become available. The NCCN Guidelines® are a consensus statement of its authors regarding their views of currently accepted approaches to treatment. Any clinician seeking to apply or consult any NCCN Guidelines® is expected to use independent medical judgment in the context of individual clinical circumstances to determine any patient’s care or treatment. The NCCN makes no warranties of any kind whatsoever regarding their content, use, or application and disclaims any responsibility for their application or use in any way.

Ocular side effects of systemically administered chemotherapy

Tables of Possible Side Effects for Commonly-Used Oncology Drugs

"The overall contribution of curative and adjuvant cytotoxic chemotherapy to 5-year survival in adults was estimated to be 2.1% in the USA . It is clear that cytotoxic chemotherapy only makes a minor contribution to cancer survival." - Graeme Morgan, Robyn Ward, and Michael Barton

2006 Chemotherapy impairs the brain, killing crucial neural cells and causing key parts of the brain to shrink.

New findings add to a growing body of evidence suggesting that the mental fuzziness, memory loss and cognitive impairment often reported by cancer patients but often dismissed by oncologists - is a serious problem.

Key areas of the brain including the prefrontal, parahippocampus and cingulate gyri shrink during chemotherapy.

The damage continues for several months after chemotherapy is stopped.

MRI scans show cause of chemobrain


"Nearly every chemotherapy patient experiences short term problems with memory and concentration.

But about 15% suffer prolonged effects of what is known medically as chemotherapy induced cognitive impairment.

The symptoms are remarkably consistent:

a mental fogginess that may include problems with memory, word retrieval, concentration, processing numbers, following instructions, multitasking and setting priorities." - Jane E. Brody

"Those of us on the front lines have known this for a long time, now we have neuropathological evidence that what we are seeing involves an anatomic change." - Dr. Stewart Fleishman*, director of cancer supportive services at Beth Israel Medical Center and St. Luke's-Roosevelt Hospital Center



chemical warfare

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